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Researchers at the University of Texas Medical Branch in Galveston  have found a way to partially avert neuropathic pain. This was done by reducing the biochemical assault by reactive oxygen species which are generated in the wake of nerve injury to keep GABA neurons could alive and healthy after peripheral nerve or tissue injury.

“GABA neurons are particularly susceptible to oxidative stress, and we hypothesized that reactive oxygen species contribute to neuropathic sensitization by promoting the loss of GABA neurons as well as hindering GABA functions,” said UTMB professor Jin Mo Chung. ”So by giving the antioxidant we lowered the pain behavior”.  Chung believes anti-oxidants have great potential as a treatment for neuropathic pain.